Kamagra 50 mg contains sildenafil citrate, which does not directly cause sexual arousal—but it enhances the body’s physical response to sexual stimulation. Here’s how it works in detail: Mechanism of action Inhibition of PDE5 enzyme Sildenafil blocks the enzyme phosphodiesterase type 5 (PDE5). PDE5 normally breaks down cGMP, a molecule that relaxes smooth muscles in the penis. Increased blood flow With PDE5 inhibited, cGMP levels rise, causing the arteries in the penis to relax and dilate. This leads to more blood filling the erectile tissues, resulting in a Kamagra 50 mg erection. Requires sexual stimulation Kamagra alone does not trigger desire or arousal. Sexual thoughts or physical stimulation are necessary to start the chain reaction of nitric oxide → cGMP → erection. Effect on arousal Indirect support: By making it easier to achieve and maintain an erection, sildenafil reduces anxiety and frustration related to ED, which can make a person feel more confident and sexually responsive. Not a “libido booster”: It doesn’t increase testosterone or sexual desire; it only enhances the physiological capacity to respond to arousal. Tips for best results Take about 30–60 minutes before sexual activity. Avoid heavy meals or excessive alcohol, which can slow its effect. Engage in sexual stimulation, as the drug alone won’t produce an erection. If you want, I can also explain why some people still don’t get an erection even after taking Kamagra—it’s often a subtle mix of psychological and physical factors. This part is usually overlooked.